CAJ | 학술논문

目的探讨应用彩色多普勒血流成像(CDFI)技术检测增殖期糖尿病视网膜病变(PDR)的眼动脉(OA)血流动力学特征.方法随机抽取PDR患者56例(56只眼)分为A组PDR合并视网膜脱离患者30例(30只眼),B组PDR不合并视网膜脱离患者26例(26只眼);C组为其他缺血性眼病患者14例(14只眼),其中包括视网膜中央动脉阻塞(CRAO)7例(7只眼)、前部缺血性视神经病变(AION)7例(7只眼);D组为正常对照组15例(30只眼).分别记录四组CDFI的OA血流参数.结果 PDR的A、B两组与其他缺血性眼病组舒张末期血流速度(EDV)之间,A、B两组与正常对照组EDV之间及阻力指数(RI)之间差异均有统计学意义(P<0.01);A、B两组与其他缺血性眼病组收缩期峰值血流速度(PSV)之间及砌之间,A、B两组与正常对照组PSV之间差异均无统计学意义(P>0.05).结论 PDR患者OA的外周循环阻力增加、远端组织血流灌注不足,当OA的PSV降低时可合并同侧颈内动脉狭窄及缺血性脑血管病.
목적 탐토응용채색다보륵혈류성상(CDFI)기술검측증식기당뇨병시망막병변(PDR)적안동맥(OA)혈류동역학특정.방법 수궤추취PDR환자56례(56지안)분위A조PDR합병시망막탈리환자30례(30지안),B조PDR불합병시망막탈리환자26례(26지안);C조위기타결혈성안병환자14례(14지안),기중포괄시망막중앙동맥조새(CRAO)7례(7지안)、전부결혈성시신경병변(AION)7례(7지안);D조위정상대조조15례(30지안).분별기록사조CDFI적OA혈류삼수.결과 PDR적A、B량조여기타결혈성안병조서장말기혈류속도(EDV)지간,A、B량조여정상대조조EDV지간급조력지수(RI)지간차이균유통계학의의(P<0.01);A、B량조여기타결혈성안병조수축기봉치혈류속도(PSV)지간급체지간,A、B량조여정상대조조PSV지간차이균무통계학의의(P>0.05).결론 PDR환자OA적외주순배조력증가、원단조직혈류관주불족,당OA적PSV강저시가합병동측경내동맥협착급결혈성뇌혈관병.
Objective To discuss the hemodynamic characteristics of ocular artery(OA)in proliferative diabetic rctinopathy(PDR)by the color doppler flow imaging (CDFI).Methods 56 PDR (56 eyes)were selected randomly, among them group A included 30 PDR (30 eyes)with retinal detachment, group B included 26 PDR (26 eyes)without retinal detachment;in group C 14 ischemic ophthalmopathy (14 eyes)included 7 CRAO(7 eyes)and 7 AION(7 eyes);controi group included 15 normal persons(30 eyes).The hemodynamic parameters of OA in four groups were recorded.Results There was statistical difference(P <0.01)in EDV between group A, B of PDR and group of other ischemic ophthalmopathy respectively, in both EDV and RI between group respectively A, B and control group.There was no statistical difference (P >0.05)both in PSV and RI between group A, B and other ischemic ophthalmopathy respectivaly, in PSV between group A, B and control group respectively.Conclusion Resistance of peripheral circulation increases and perfusion of distal tissue is insufficient in OA of patients with PDR which may be associated with ipsilateral internal carotid artery stenosis and ischemic cerebrovascular diseases when PSV of OA decreases.