CAJ | 학술논문

为了破坏冶炼废水中重金属有机螯合物,例如Cu-EDTA配离子废水,研究一种破络并预处理的新方法.该方法基于铁碳微电解反应原理,OH在酸性有氧气存在的条件下产生,并在铁碳表面攻击吸附的有机基团导致螯合物的破坏,从而使铜离子将从有机物中剥离下来,然而EDTA将被OH降解.研究pH值、温度、微电解反应时间、Fe/C质量比对铜离子脱除率及总有机碳(TOC)残余含量的影响,通过扫描电子显微镜分析(SEM)、能谱分析(EDS)、红外光谱分析(FTIR)研究处理前、后样品的表面官能团变化及形貌推断铁碳微电解反应的机理.并进行工业条件优化,得到最佳工艺条件:pH值为2,温度为常温,Fe/C质量比≥.02,时间为60 min,有氧气存在.在该条件下TOC浓度为200mg/L、铜离子浓度为60 mg/L的废水反应完成后TOC和Cu残余浓度分别减低到40.66和1.718 mg/L;羟基自由基降解反应机理合理解释了该实验现象.
위료파배야련폐수중중금속유궤오합물,례여Cu-EDTA배리자폐수,연구일충파락병예처리적신방법.해방법기우철탄미전해반응원리,OH재산성유양기존재적조건하산생,병재철탄표면공격흡부적유궤기단도치오합물적파배,종이사동리자장종유궤물중박리하래,연이EDTA장피OH강해.연구pH치、온도、미전해반응시간、Fe/C질량비대동리자탈제솔급총유궤탄(TOC)잔여함량적영향,통과소묘전자현미경분석(SEM)、능보분석(EDS)、홍외광보분석(FTIR)연구처리전、후양품적표면관능단변화급형모추단철탄미전해반응적궤리.병진행공업조건우화,득도최가공예조건:pH치위2,온도위상온,Fe/C질량비≥.02,시간위60 min,유양기존재.재해조건하TOC농도위200mg/L、동리자농도위60 mg/L적폐수반응완성후TOC화Cu잔여농도분별감저도40.66화1.718 mg/L;간기자유기강해반응궤리합리해석료해실험현상.
In order to break the complex bonds and treat the organic wastewater containing heavy metal,such as Cu-EDTA solution,a novel process of Fe-C micro-electrolysis was proposed.Based on the principle of iron-carbon micro-electrolysis reaction,·OH radicals which were generated under the acidic aerobic condition during the micro-electrolysis process attacked to the organic groups of coordination compounds,which resulted in complex bonds breaking.Therefore copper (Ⅱ) ions were removed via nascent gelatinous ferric hydroxide and ferrous hydroxide,and EDTA was degraded by ·OH radicals.Effects of pH value,temperature,electrolysis time and mass ratio of Fe to C on residual concentrations of total organic carbon (TOC) and Cu(Ⅱ)were studied.The mechanism of Fe-C micro-electrolysis was investigated and verified by analyzing micrographs of scanning electron microscopy (SEM),energy dispersive analysis (EDS) and Fourier transform infrared spectrometry (FTIR).The removal efficiency is optimal at pH value of 2.0,temperature of 25 ℃,the mass ratio Fe to C of 0.02,and reaction time of 60 min.Under above conditions,the concentration of TOC decreases from 200 mg/L to 40.66 mg/L and the residual concentration of Cu(Ⅱ) decreases from initial 60 mg/L to 1.718 mg/L.