国际儿科学杂志
國際兒科學雜誌
국제인과학잡지
INTERNATIONAL JOURNAL OF PEDIATRICS
2010年
6期
574-576
,共3页
炎症反应%高氧肺损伤%细胞因子%核转录因子-κB
炎癥反應%高氧肺損傷%細胞因子%覈轉錄因子-κB
염증반응%고양폐손상%세포인자%핵전록인자-κB
Inflammation%Hyperoxia-induced lung injury%Cytokines%Nuclear factor - kappa B
动物实验及临床研究均证明炎症反应参与了高氧肺损伤的过程,但炎症本身及其对高氧肺损伤的影响相当复杂.高氧肺损伤后,多种细胞因子表达及炎细胞浸润构成了高氧肺损伤向炎症性损伤转变的基础.
動物實驗及臨床研究均證明炎癥反應參與瞭高氧肺損傷的過程,但炎癥本身及其對高氧肺損傷的影響相噹複雜.高氧肺損傷後,多種細胞因子錶達及炎細胞浸潤構成瞭高氧肺損傷嚮炎癥性損傷轉變的基礎.
동물실험급림상연구균증명염증반응삼여료고양폐손상적과정,단염증본신급기대고양폐손상적영향상당복잡.고양폐손상후,다충세포인자표체급염세포침윤구성료고양폐손상향염증성손상전변적기출.
Animal experiments and clinical studies both support the inflammatory response involved in the process of hyperoxia-induced lung injury,but the inflammation and its influence is fairly complicated. A variety of cytokines expression and inflammatory cells infiltration in hyperoxic lung injury form the basis of changes in inflammatory injury.
