中国医药
中國醫藥
중국의약
CHINA MEDICINE
2010年
9期
842-843
,共2页
糖尿病,2型%交感神经系统%脑血管意外%自主神经系统疾病
糖尿病,2型%交感神經繫統%腦血管意外%自主神經繫統疾病
당뇨병,2형%교감신경계통%뇌혈관의외%자주신경계통질병
Diabetes mellitus,type 2%Sympathetic nervous system%Cerebrovascular accident%Autonomic nervous system diseases
目的 探讨交感神经皮肤反应(SSR)检测在评价2型糖尿病合并脑卒中患者自主神经损害中的价值.方法 对100例2型糖尿病合并脑卒中患者进行SSR检测,另选取50例脑卒中患者和40例糖尿病患者作为对照.结果 糖尿病合并脑卒中组平均起始潜伏期(1.76±1.49)s,平均波幅(1.45±0.81)mV;脑卒中组分别为(1.46±1.26)s、(2.29±0.41)mV;糖尿病组分别为(1.48±1.29)s、(2.32±0.44)mV.糖尿病合并脑卒中组与后两组SSR的潜伏期、波幅比较差异有统计学意义(P＜0.01).70%的脑卒中患者和60%的糖尿病患者可引出SSR,而糖尿病合并脑卒中患者仅15%可正常引出SSR,85%患者至少有一侧肢体SSR异常,表现为潜伏期延长、波幅降低、甚至波形消失,且下肢异常率高于上肢(P＜0.05).结论 SSR可作为评价糖尿病合并脑卒中患者自主神经功能的一项简便易行、安全无创的客观检查方法.
目的 探討交感神經皮膚反應(SSR)檢測在評價2型糖尿病閤併腦卒中患者自主神經損害中的價值.方法 對100例2型糖尿病閤併腦卒中患者進行SSR檢測,另選取50例腦卒中患者和40例糖尿病患者作為對照.結果 糖尿病閤併腦卒中組平均起始潛伏期(1.76±1.49)s,平均波幅(1.45±0.81)mV;腦卒中組分彆為(1.46±1.26)s、(2.29±0.41)mV;糖尿病組分彆為(1.48±1.29)s、(2.32±0.44)mV.糖尿病閤併腦卒中組與後兩組SSR的潛伏期、波幅比較差異有統計學意義(P＜0.01).70%的腦卒中患者和60%的糖尿病患者可引齣SSR,而糖尿病閤併腦卒中患者僅15%可正常引齣SSR,85%患者至少有一側肢體SSR異常,錶現為潛伏期延長、波幅降低、甚至波形消失,且下肢異常率高于上肢(P＜0.05).結論 SSR可作為評價糖尿病閤併腦卒中患者自主神經功能的一項簡便易行、安全無創的客觀檢查方法.
목적 탐토교감신경피부반응(SSR)검측재평개2형당뇨병합병뇌졸중환자자주신경손해중적개치.방법 대100례2형당뇨병합병뇌졸중환자진행SSR검측,령선취50례뇌졸중환자화40례당뇨병환자작위대조.결과 당뇨병합병뇌졸중조평균기시잠복기(1.76±1.49)s,평균파폭(1.45±0.81)mV;뇌졸중조분별위(1.46±1.26)s、(2.29±0.41)mV;당뇨병조분별위(1.48±1.29)s、(2.32±0.44)mV.당뇨병합병뇌졸중조여후량조SSR적잠복기、파폭비교차이유통계학의의(P＜0.01).70%적뇌졸중환자화60%적당뇨병환자가인출SSR,이당뇨병합병뇌졸중환자부15%가정상인출SSR,85%환자지소유일측지체SSR이상,표현위잠복기연장、파폭강저、심지파형소실,차하지이상솔고우상지(P＜0.05).결론 SSR가작위평개당뇨병합병뇌졸중환자자주신경공능적일항간편역행、안전무창적객관검사방법.
Objective To investigate the significance of sympathetic skin response (SSR) to automatic dysfunction in patients with type 2 diabetes mellitus (T2DM) combined with cerebral infarction. Methods SSR was carried out in 100 patients with T2DM combined with cerebral infarction and 50 patients with cerebral infarction and 40 patients with diabetes were enrolled as control groups. Results There was significant difference of latencies and amplitudes of SSR between three groups (P <0.01). SSR was elicited clearly in 70 percent cerebral infarction and 60 percent diabetes controls, but SSR was elicited clearly in only 15 patients with T2DM combined with cerebral infarction. 85 patients had abnormal responses in SSR in at least one limb, and showed prolongation of latency and remarkable decreases in amplitude. Conclusions SSR is an objective diagnostic method to assess the autonomic nerve dysfunction related to T2DM combined with cerebral infarction.
