中国临床实用医学
中國臨床實用醫學
중국림상실용의학
CHINA CLINICAL PRACTICAL MEDICINE
2010年
2期
116-117
,共2页
程宏松%张有亭%胡宏阁%赵晏黎
程宏鬆%張有亭%鬍宏閣%趙晏黎
정굉송%장유정%호굉각%조안려
青光眼%睫状环阻滞%药物%手术
青光眼%睫狀環阻滯%藥物%手術
청광안%첩상배조체%약물%수술
Glaucoma%Ciliary-block%Drug%Operation
目的 分析药物性睫状环阻滞性青光眼的临床特点,总结其治疗方法和治疗效果.方法 对2001年4月至2008年12月在本院治疗的此类型睫状环阻滞性青光眼的临床资料进行总结、回顾性分析.结果 ①眼压:入院时的眼压43.38～81.78 mm Hg,平均为(51.66±5.46)mm Hg;经治疗10～14 d后,所有患者眼压均控制在25 mm Hg以内.随访12月后,2例长期局部点眼药眼压一直维持在20 mm Hg左右,其他采用手术治疗的10例患者,有6例不用降眼压药物,眼压控制在20 mm Hg以内;3例眼压在21～25 mm Hg,给予局部点β-受体阻滞剂,眼压控制在20 mm Hg以内,1例眼压失控而再次手术,最终失明;②视力:治疗前12例最佳矫正视力为1 m光感至0.3,治疗后最佳矫正视力0.1～0.8.治疗前后视力比较,差异有显著性(P<0.01).结论 这类睫状环阻滞性青光眼都具有闭角型青光眼的解剖结构,用毛果芸香碱眼压增高,用眼压下降,积极治疗可恢复有用视力.
目的 分析藥物性睫狀環阻滯性青光眼的臨床特點,總結其治療方法和治療效果.方法 對2001年4月至2008年12月在本院治療的此類型睫狀環阻滯性青光眼的臨床資料進行總結、迴顧性分析.結果 ①眼壓:入院時的眼壓43.38～81.78 mm Hg,平均為(51.66±5.46)mm Hg;經治療10～14 d後,所有患者眼壓均控製在25 mm Hg以內.隨訪12月後,2例長期跼部點眼藥眼壓一直維持在20 mm Hg左右,其他採用手術治療的10例患者,有6例不用降眼壓藥物,眼壓控製在20 mm Hg以內;3例眼壓在21～25 mm Hg,給予跼部點β-受體阻滯劑,眼壓控製在20 mm Hg以內,1例眼壓失控而再次手術,最終失明;②視力:治療前12例最佳矯正視力為1 m光感至0.3,治療後最佳矯正視力0.1～0.8.治療前後視力比較,差異有顯著性(P<0.01).結論 這類睫狀環阻滯性青光眼都具有閉角型青光眼的解剖結構,用毛果蕓香堿眼壓增高,用眼壓下降,積極治療可恢複有用視力.
목적 분석약물성첩상배조체성청광안적림상특점,총결기치료방법화치료효과.방법 대2001년4월지2008년12월재본원치료적차류형첩상배조체성청광안적림상자료진행총결、회고성분석.결과 ①안압:입원시적안압43.38～81.78 mm Hg,평균위(51.66±5.46)mm Hg;경치료10～14 d후,소유환자안압균공제재25 mm Hg이내.수방12월후,2례장기국부점안약안압일직유지재20 mm Hg좌우,기타채용수술치료적10례환자,유6례불용강안압약물,안압공제재20 mm Hg이내;3례안압재21～25 mm Hg,급여국부점β-수체조체제,안압공제재20 mm Hg이내,1례안압실공이재차수술,최종실명;②시력:치료전12례최가교정시력위1 m광감지0.3,치료후최가교정시력0.1～0.8.치료전후시력비교,차이유현저성(P<0.01).결론 저류첩상배조체성청광안도구유폐각형청광안적해부결구,용모과예향감안압증고,용안압하강,적겁치료가회복유용시력.
Objective To study clinical characteristic of drug causingciliary-block glaucoma,to evaluate the therapeutic effect of surgery for drug causing ciliary-block glaucoma. Methods The clinical informations of 12cases( 12 eyes) of drug causing ciliary-block glaucoma diagnosed in our hospital between April 2001 and December 2008 were evaluated, vistaed summary. Results ①Intraocular pressure (IOP) :Being hospitalized,the IOP of patients was 43.38 ～ 81.78 mm Hg, the mean was (51.66 ± 5.46 ) mm Hg; After treating for 10 ～ 14 days, the IOP of 12 patients were controlled below 25 mm Hg. Following up 12 months, two patients kept their IOP at about 20 mm Hg by using eye-drops; The other 10 patients performed an operation, the IOP of 6 patients were controlled at about 20 mm Hg without giving medicine; Three patients were 21 ～ 25 mm Hg, their IOP was
controlled at 20 mm Hg after giving β-receptor block medicine. The IOP of one patient was out of control; the patient lost his eye' s functioning after performing the operation. ②Vision: Before treating patients, they had light sense or vision up to 0. 3 in one meter distance ( after being corrected defects of vision). After treating patients,the best-corrected defects of vision were 0. 1 ～ 0. 8. There was a significant difference in vision between before and after treating patients. Conclusion Ciliary-block glaucoma caused by medicine has the same anatomy features as PACG.; The IOP going up was caused by Plocarpine ,The IOP going down was caused by Tropicamidum or Atropine; Vision can be restored through vigorous treating.
