CAJ | 학술논문

目的观察血糖控制良好的2型糖尿病患者静息能量消耗(REE)的特点.方法采用代谢车测量45例血糖稳定的2型糖尿病患者的REE,记录其一般情况、生化指标、Harris-Benedict和Owen公式计算基础能量消耗预测值,并进行比较分析.结果血糖稳定的2型糖尿病患者的静息能量消耗实测值与Harris-Benedict及Owen公式预测值比较差异无统计学意义(P＞0.05),相关分析显示REE与性别、年龄、体重、身高、体表面积、去脂组织显著相关(P＜0.05),与体重指数、脂肪组织、空腹血糖、餐后2 h血糖、糖化血红蛋白、总胆固醇、三酰甘油、总蛋白、白蛋白、血红蛋白无相关性(P＞0.05),多元逐步回归分析显示去脂组织和年龄与REE关系最密切.结论血糖良好的2 型糖尿病患者的REE并不增高,决定其REE的因素与健康个体相似,测定REE能更好地指导糖尿病的医学营养治疗.
목적 관찰혈당공제량호적2형당뇨병환자정식능량소모(REE)적특점.방법 채용대사차측량45례혈당은정적2형당뇨병환자적REE,기록기일반정황、생화지표、Harris-Benedict화Owen공식계산기출능량소모예측치,병진행비교분석.결과 혈당은정적2형당뇨병환자적정식능량소모실측치여Harris-Benedict급Owen공식예측치비교차이무통계학의의(P＞0.05),상관분석현시REE여성별、년령、체중、신고、체표면적、거지조직현저상관(P＜0.05),여체중지수、지방조직、공복혈당、찬후2 h혈당、당화혈홍단백、총담고순、삼선감유、총단백、백단백、혈홍단백무상관성(P＞0.05),다원축보회귀분석현시거지조직화년령여REE관계최밀절.결론 혈당량호적2 형당뇨병환자적REE병불증고,결정기REE적인소여건강개체상사,측정REE능경호지지도당뇨병적의학영양치료.
Objective To investigate the features of resting energy expenditure (REE) in patients with well controlled type 2 diabetes mellitus (T2DM). Methods Totally 45 T2DM patients with stable blood glucose were enrolled. The general conditions, biochemical indicators, measurements of REE ( MREE), and basal energy expenditure (BEE) calculated with Harris-Benedict formula (HBEE) and Owen formula (OBEE) were recorded and compared. Results MREE had no significant difference with HBEE or OBEE in T2DM patients with stable blood glucose (P ＞ 0. 05). Correlation analysis showed that REE was significantly correlated with gender, age, body weight, body height, body surface area, and fat-free mass ( all P ＜ 0.05 ), but was not correlated with body mass index, fat mass, fasting plasma glucose, postprandial plasma glucose, haemoglobin Alc, total cholesterol, triglyceride, total protein, albumin, and haemoglobin (P ＞ 0. 05 ). Multiple regression analysis showed that fat free mass and age had the closest correlation with REE. Conclusions REE does not increase in T2DM patients with well controlled blood glucose. Factors that influence their REE are similar with healthy individuals. Determi nation of REE can provide useful information for the nutrition treatment of T2DM.