CAJ | 학술논문

突触前和突触后细胞内钙离子（［Ca2+］i）在短时程和长时程突触的可塑性中，发挥着重要的信息传递作用。兴奋后残留［Ca2+］i ，可以激发短时程突触增强。突触前［Ca2+］i可以影响被抑制的突触前膜囊泡的更新，并准确编码突触前和突触后信息，产生截然相反的长时程突触修饰(LTP或LTD)。
돌촉전화돌촉후세포내개리자（［Ca2+］i）재단시정화장시 정돌촉적가소성중，발휘착중요적신식전체작용。흥강후잔류［Ca2+］i ，가이 격발단시정돌촉증강。돌촉전［Ca2+］i가이영향피억제적돌촉전막낭포적경신， 병준학편마돌촉전화돌촉후신식，산생절연상반적장시정돌촉수식(LTP혹LTD)。
Recent work shows that the intracellular free Ca2＋ concentrations (［Ca2＋］i) of the presynaptic and postsynaptic neurons play crucial signaling roles in short- and lon g-term synaptic plasticit y. Residual ［Ca2+］i followed conditioning stimulation may cause short -term synapt ic enhancement. Presynaptic ［Ca2+］i could influence the replacing of presynaptic depressed vesicles, as well as encode the precise relative timing of presynaptic input and postsynaptic activity and generate long-term synaptic modification o f opposite polarity(LTP or LTD).