中华胰腺病杂志
中華胰腺病雜誌
중화이선병잡지
CHINESE JOURNAL OF PANCREATOLOGY
2010年
3期
174-176
,共3页
马天顺%卢明智%邵成伟%左长京%陆建平%吕桃珍%郑建明
馬天順%盧明智%邵成偉%左長京%陸建平%呂桃珍%鄭建明
마천순%로명지%소성위%좌장경%륙건평%려도진%정건명
胰腺肿瘤%体层摄影术,X线计算机%诊断,鉴别%癌,非典型
胰腺腫瘤%體層攝影術,X線計算機%診斷,鑒彆%癌,非典型
이선종류%체층섭영술,X선계산궤%진단,감별%암,비전형
Pancreatic neoplasms%Tomngraphy,X-ray computed%Diagnosis,differential%Cancer,atypical
目的 分析胰腺癌的64排螺旋CT的不典型表现,以提高对该肿瘤的CT征象的认识水平.方法 回顾性分析经手术病理证实的缺乏典型CT征象的12例胰腺导管腺癌的64排螺旋CT资料.结果 12例均为胰腺导管腺癌.其中,中分化导管腺癌7例,中高分化导管腺癌1例;黏液腺癌1例;腺鳞癌3例.8例导管腺癌病灶中位于胰头及(或)钩突部7例,胰颈部1例,表现为等、低密度或囊实性肿块,增强后无明显强化;5例肿瘤呈明显外生性或有外生倾向;5例肿瘤远端胰管无扩张,2例出现胆总管和肝内胆管扩张,仅1例出现肿瘤远端胰腺萎缩.1例黏液腺癌CT平扫示胰头部5 cm囊性病灶,增强后仅囊性病灶下方少许实性部分轻度强化,体尾部胰管中度扩张(7 mmn),胆总管及邻近血管未受侵犯.3例腺鳞癌病灶中位于胰头2例,胰体部1例,肿块最大径3.0~4.5 cm,CT增强扫描胰腺实质期示3例病灶内均见液化坏死区,病灶远端胰管均轻度扩张(4~5 mm),胆总管和肝内胆管均未见扩张.结论 胰腺癌可出现不典型的CT影像表现,要注意与易混淆疾病进行鉴别诊断.
目的 分析胰腺癌的64排螺鏇CT的不典型錶現,以提高對該腫瘤的CT徵象的認識水平.方法 迴顧性分析經手術病理證實的缺乏典型CT徵象的12例胰腺導管腺癌的64排螺鏇CT資料.結果 12例均為胰腺導管腺癌.其中,中分化導管腺癌7例,中高分化導管腺癌1例;黏液腺癌1例;腺鱗癌3例.8例導管腺癌病竈中位于胰頭及(或)鉤突部7例,胰頸部1例,錶現為等、低密度或囊實性腫塊,增彊後無明顯彊化;5例腫瘤呈明顯外生性或有外生傾嚮;5例腫瘤遠耑胰管無擴張,2例齣現膽總管和肝內膽管擴張,僅1例齣現腫瘤遠耑胰腺萎縮.1例黏液腺癌CT平掃示胰頭部5 cm囊性病竈,增彊後僅囊性病竈下方少許實性部分輕度彊化,體尾部胰管中度擴張(7 mmn),膽總管及鄰近血管未受侵犯.3例腺鱗癌病竈中位于胰頭2例,胰體部1例,腫塊最大徑3.0~4.5 cm,CT增彊掃描胰腺實質期示3例病竈內均見液化壞死區,病竈遠耑胰管均輕度擴張(4~5 mm),膽總管和肝內膽管均未見擴張.結論 胰腺癌可齣現不典型的CT影像錶現,要註意與易混淆疾病進行鑒彆診斷.
목적 분석이선암적64배라선CT적불전형표현,이제고대해종류적CT정상적인식수평.방법 회고성분석경수술병리증실적결핍전형CT정상적12례이선도관선암적64배라선CT자료.결과 12례균위이선도관선암.기중,중분화도관선암7례,중고분화도관선암1례;점액선암1례;선린암3례.8례도관선암병조중위우이두급(혹)구돌부7례,이경부1례,표현위등、저밀도혹낭실성종괴,증강후무명현강화;5례종류정명현외생성혹유외생경향;5례종류원단이관무확장,2례출현담총관화간내담관확장,부1례출현종류원단이선위축.1례점액선암CT평소시이두부5 cm낭성병조,증강후부낭성병조하방소허실성부분경도강화,체미부이관중도확장(7 mmn),담총관급린근혈관미수침범.3례선린암병조중위우이두2례,이체부1례,종괴최대경3.0~4.5 cm,CT증강소묘이선실질기시3례병조내균견액화배사구,병조원단이관균경도확장(4~5 mm),담총관화간내담관균미견확장.결론 이선암가출현불전형적CT영상표현,요주의여역혼효질병진행감별진단.
Objective To analyze atypical 64-slice spiral CT imaging finings of pancreatic cancer and to improve the ability to identify CT manifestations of pancreatic cancer. Methods A retrospective analysis was performed on the atypical 64-slice spiral CT imaging findings of 12 eases of pancreatic cancer confirmed by pathology after surgery. Results All the twelve cases were pancreatic ductal adenocarcinoma.Among them, 7 cases were moderately differentiated ductal adenocarcinoma, 1 case was well-differentiated ductal adenocarcinoma, 1 case was mucinous adenocarcinoma, 3 cases were adenosquamous carcinoma. Among 8 cases with ductal adenocarcinoma, the lesions were located in the pancreatic head and (or) uncinate process in 7 cases, and in the pancreatic neck of 1 case. Tumors were expressed as isodense or low-density or cysticsolid lesions, the masses showed no enhancement in the enhanced scanning phase. Tumors were clearly exogenous or exogenous tendencies in 5 cases. Five cases had no distal pancreatic duct dilation, 2 patients had common bile duct and intrahepatic biliary dilation, and only 1 patient had atrophy of distal pancreas. There was one case of mucinous carcinoma, plain CT scan showed a cystic lesion in head of pancreas about 5cm in diameter, the solid part below the cystic lesion was slightly enhamced in the enhanced scanning phase and the body and tail pancreatic duct was moderately dilated (7 mm). There was no common bile duct and adjacent blood vessels invasion. Among 3 cases of adenosquamous carcinoma, lesions were located in the pancreatic head of 2 cases and in pancreatic body of 1 case. The maximal diameter of mass ranged 3.0 cm ~ 4.5 cm.Cystic necrotic area was observed within the lesions in 3 cases in enhanced pancreatic parenchymal phase of CT scan. Distal pancreatic duct were mildly dilated (4 ~ 5 mm) in 3 cases. There was no common bile duct and intrahepatic bile duct dilation. Conclusions Pancreatic cancer may show atypical CT imaging findings and great cautions are needed for differential diagnosis.