CAJ | 학술논문

肺炎支原体经飞沫传播，侵入呼吸道黏膜后，通过其特殊的结构、紧密吸附于易感宿主的细胞膜的受体上，在其表现增殖并释放毒性物质，如过氧化氢、酶、膜脂类等，造成组织损伤。其基本病理改变是间质性肺炎及急性毛细支气管炎，显微镜下可见局部黏膜组织充血、水肿、变厚，细胞膜被损伤，上皮细胞纤毛运动消失，单核细胞及浆细胞浸润，细支气管中可见到中性粒细胞及坏死的上皮细胞。迅速、积极地控制感染是治疗肺炎支原体肺炎的重要环节。布地奈德混悬液是一种经喷射雾化吸入的糖皮质激素，有较高的糖皮质激素受体结合力，能抑制呼吸道炎性细胞及介质释放，减轻呼吸道高敏反应，减少黏膜水肿及黏液分泌，修复呼吸道，迅速缓解临床症状。
폐염지원체경비말전파，침입호흡도점막후，통과기특수적결구、긴밀흡부우역감숙주적세포막적수체상，재기표현증식병석방독성물질，여과양화경、매、막지류등，조성조직손상。기기본병리개변시간질성폐염급급성모세지기관염，현미경하가견국부점막조직충혈、수종、변후，세포막피손상，상피세포섬모운동소실，단핵세포급장세포침윤，세지기관중가견도중성립세포급배사적상피세포。신속、적겁지공제감염시치료폐염지원체폐염적중요배절。포지내덕혼현액시일충경분사무화흡입적당피질격소，유교고적당피질격소수체결합력，능억제호흡도염성세포급개질석방，감경호흡도고민반응，감소점막수종급점액분비，수복호흡도，신속완해림상증상。
mycoplasma pneumoniae spread by droplets, after respiratory tract mucous membrane, by its special structure, closely adsorbed on the susceptibility of the host cell membrane receptor, performance in its proliferation and releasing toxic substances, such as hydrogen peroxide, enzymes, membrane lipid, result in tissue damage. The basic pathological change is interstitial pneumonia and acute capillary bronchitis, microscopi-cally visible local mucosal hyperemia, edema and thickening, the cell membrane is broken, epithelial cilia movement disappeared, mononuclear cells and plasma cell infiltrates, neutrophils are seen in the bronchioles and necrosis of epithelial cells. Quickly and actively controlling infection is an im-portant part of the treatment of mycoplasma pneumoniae pneumonia. Budesonide suspension liquid is a kind of the spray atomization inhalation of glu-cocorticoid, have higher glucocorticoid receptor binding force, can inhibit airway inflammatory cells and media release, reduce respiratory gao min re-action, reduce mucosal edema and mucus secretion, repair of the respiratory tract, quickly relieve clinical symptoms.