四川医学
四川醫學
사천의학
SICHUAN MEDICAL JOURNAL
2014年
7期
868-870,871
,共4页
张玉梅%余朝萍%许锋成%刘颖%曾洪燕%郭华莉%晏明君%刘于菲%刘天虎
張玉梅%餘朝萍%許鋒成%劉穎%曾洪燕%郭華莉%晏明君%劉于菲%劉天虎
장옥매%여조평%허봉성%류영%증홍연%곽화리%안명군%류우비%류천호
超声技术血管%回声跟踪技术%吸烟%颈动脉%动脉硬化
超聲技術血管%迴聲跟蹤技術%吸煙%頸動脈%動脈硬化
초성기술혈관%회성근종기술%흡연%경동맥%동맥경화
ultrasound%echo-tracking%smoking%carotid arteries%arteriosclerosis
目的:利用超声技术及血管回声跟踪技术(Echo tracking technology,ET)评价男性吸烟者颈动脉弹性功能。方法将符合要求的男性受试者,吸烟者112例,非吸烟者80例,检测各组颈总动脉内中膜厚度(Intima-media thickness, IMT)、并分析动脉硬化参数,包括动脉僵硬系数(Arterial stiffness coefficient,β)、压力-应变弹性系数(Pressure elastic coeffi-cient, Ep)、动脉顺应性(Artery compliance,AC)、膨大指数(Adamkiewicz index, AI)及脉搏波传导速度(Pulse wave velocity, PWV)。结果男性吸烟者双侧颈总动脉窦部和左侧颈总动脉 IMT 较男性非吸烟者增厚,差异有统计学意义,动脉僵硬系数(β)、压力-应变弹性系数(Ep)、脉搏波传导速度(PWV)增加,膨大指数(AI)及动脉顺应性(AC)下降。结论长期大量吸烟可以导致脂代谢紊乱,并可导致颈总动脉内中膜厚度增加,损伤血管内皮功能导致动脉硬化。
目的:利用超聲技術及血管迴聲跟蹤技術(Echo tracking technology,ET)評價男性吸煙者頸動脈彈性功能。方法將符閤要求的男性受試者,吸煙者112例,非吸煙者80例,檢測各組頸總動脈內中膜厚度(Intima-media thickness, IMT)、併分析動脈硬化參數,包括動脈僵硬繫數(Arterial stiffness coefficient,β)、壓力-應變彈性繫數(Pressure elastic coeffi-cient, Ep)、動脈順應性(Artery compliance,AC)、膨大指數(Adamkiewicz index, AI)及脈搏波傳導速度(Pulse wave velocity, PWV)。結果男性吸煙者雙側頸總動脈竇部和左側頸總動脈 IMT 較男性非吸煙者增厚,差異有統計學意義,動脈僵硬繫數(β)、壓力-應變彈性繫數(Ep)、脈搏波傳導速度(PWV)增加,膨大指數(AI)及動脈順應性(AC)下降。結論長期大量吸煙可以導緻脂代謝紊亂,併可導緻頸總動脈內中膜厚度增加,損傷血管內皮功能導緻動脈硬化。
목적:이용초성기술급혈관회성근종기술(Echo tracking technology,ET)평개남성흡연자경동맥탄성공능。방법장부합요구적남성수시자,흡연자112례,비흡연자80례,검측각조경총동맥내중막후도(Intima-media thickness, IMT)、병분석동맥경화삼수,포괄동맥강경계수(Arterial stiffness coefficient,β)、압력-응변탄성계수(Pressure elastic coeffi-cient, Ep)、동맥순응성(Artery compliance,AC)、팽대지수(Adamkiewicz index, AI)급맥박파전도속도(Pulse wave velocity, PWV)。결과남성흡연자쌍측경총동맥두부화좌측경총동맥 IMT 교남성비흡연자증후,차이유통계학의의,동맥강경계수(β)、압력-응변탄성계수(Ep)、맥박파전도속도(PWV)증가,팽대지수(AI)급동맥순응성(AC)하강。결론장기대량흡연가이도치지대사문란,병가도치경총동맥내중막후도증가,손상혈관내피공능도치동맥경화。
Objective Use of ultrasound and vascular Echo tracking technology (ET) to evaluate the effects of smoking on carotid vascular function. Methods 112 active male smokers and 80 male nonsmokers were enrolled,ultrasound and ET were used to detect the carotid vascular intima-media thickness(IMT), arterial stiffness coefficient(beta), pressure elastic coefficient (Ep), artery compliance(AC), Adamkiewicz index(AI) and pulse wave velocity(PWV). Results Compared to the male non-smokers, bilateral carotid sinus and the left common carotid artery intima-media thickness increased in active male smokers, the difference is significant. Arterial stiffness coefficient (β) rised,Pressure elastic coefficient ( Ep ) increased,Pulse wave velocity ( PWV ) accelerated, Adamkiewicz index ( AI ) and Artery compliance(AC) dropped . Conclusion long-term heavy smoking may affect the lipid, leading to abnormal metabolism of blood lipid and endothelial dysfunction, leading to arteriosclerosis.