CAJ | 학술논문

感染后肠易激综合征患者血清生化学未见异常且结肠镜检查未见形态学改变，而显微镜下却可见肠道炎症细胞浸润。肠道黏膜肥大细胞、淋巴细胞、嗜铬细胞等免疫内分泌细胞数量和功能发生改变，肠道黏膜、外周血细胞因子的异常导致肠道炎症低度反应，导致肠易激综合征的症状迁延反复。本文综合国内外有关感染后肠易激综合征的研究进展，从免疫内分泌细胞及细胞因子两个方面做一概述。
감염후장역격종합정환자혈청생화학미견이상차결장경검사미견형태학개변，이현미경하각가견장도염증세포침윤。장도점막비대세포、림파세포、기락세포등면역내분비세포수량화공능발생개변，장도점막、외주혈세포인자적이상도치장도염증저도반응，도치장역격종합정적증상천연반복。본문종합국내외유관감염후장역격종합정적연구진전，종면역내분비세포급세포인자량개방면주일개술。
There is no serum biochemistry and morphological changes in the patient of post-infectious irritable bowel syndrome, and the microscope is visible intestinal inflammatory cell infiltration. The number and function of the mast cells, lymphocytes chromaffin cell, etc change, cytokines lead to low intestinal inflammation reaction, which cause the symptoms of irritable bowel syndrome delay. The post-infectious irritable bowel syndrome are integrated in this paper from immune endocrine cells and cytokines two aspects to make a review.