中国实用医药
中國實用醫藥
중국실용의약
CHINA PRACTICAL MEDICAL
2013年
36期
1-3,4
,共4页
周龙%张建华%林进皇%俞雄杰%郁毅刚
週龍%張建華%林進皇%俞雄傑%鬱毅剛
주룡%장건화%림진황%유웅걸%욱의강
火炮打击%兔%应激防护模型%脑组织%NMDA-R1
火砲打擊%兔%應激防護模型%腦組織%NMDA-R1
화포타격%토%응격방호모형%뇌조직%NMDA-R1
Artillery attack%Rabbit%Stress protective model%Brain tissue%NMDA-R1
目的:探讨实弹演习火炮打击后,不同位置掩体保护下兔脑组织病理及NMDA-R1表达的变化。方法闽南山地团进攻演习,30只实验兔随机分为空白对照组,反角隐蔽组和迎面隐蔽组。火炮打击应激损伤后6 h,各组存活兔取脑组织,观察其病理损伤形态学及NMDA-R1表达的变化。结果与空白对照组比较,反角隐蔽组和迎面隐蔽组NMDA-R1表达强阳性,脑组织结构层次被破坏,神经细胞变性坏死,间质出血;迎面隐蔽组NMDA-R1免疫反应强度亦高于反角隐蔽组。结论火炮打击后兔脑组织应激损伤反应强烈,反角掩体保护能够减轻脑组织损伤, NMDA-R1参与脑组织损伤病理过程。
目的:探討實彈縯習火砲打擊後,不同位置掩體保護下兔腦組織病理及NMDA-R1錶達的變化。方法閩南山地糰進攻縯習,30隻實驗兔隨機分為空白對照組,反角隱蔽組和迎麵隱蔽組。火砲打擊應激損傷後6 h,各組存活兔取腦組織,觀察其病理損傷形態學及NMDA-R1錶達的變化。結果與空白對照組比較,反角隱蔽組和迎麵隱蔽組NMDA-R1錶達彊暘性,腦組織結構層次被破壞,神經細胞變性壞死,間質齣血;迎麵隱蔽組NMDA-R1免疫反應彊度亦高于反角隱蔽組。結論火砲打擊後兔腦組織應激損傷反應彊烈,反角掩體保護能夠減輕腦組織損傷, NMDA-R1參與腦組織損傷病理過程。
목적:탐토실탄연습화포타격후,불동위치엄체보호하토뇌조직병리급NMDA-R1표체적변화。방법민남산지단진공연습,30지실험토수궤분위공백대조조,반각은폐조화영면은폐조。화포타격응격손상후6 h,각조존활토취뇌조직,관찰기병리손상형태학급NMDA-R1표체적변화。결과여공백대조조비교,반각은폐조화영면은폐조NMDA-R1표체강양성,뇌조직결구층차피파배,신경세포변성배사,간질출혈;영면은폐조NMDA-R1면역반응강도역고우반각은폐조。결론화포타격후토뇌조직응격손상반응강렬,반각엄체보호능구감경뇌조직손상, NMDA-R1삼여뇌조직손상병리과정。
Objective To study the NMDA-R1 expression and pathology on injury brain tissue in rabbits hidden in different positions of blindage after real artillery attacking in battlefield. Methods 30 Japanese white rabbits were randomly divided into 3 groups, blank group, reverse hidden group and frontage hidden group in mountain regiment artillery attacking maneuver at southern of Fujian. The NMDA-R1 expression and pathology on injury brain tissue in rabbits were observed and recorded in 6 hours after injury. Results Compared with the blank group, the NMDA-R1 expression in reverse hidden group and frontage hidden group were strong positive, brain tissue structure were destroyed, nerve cells degeneration necrosis and brain tissue interstitial hemorrhage. Tthe NMDA-R1 expression in frontage hidden group were stronger than reverse hidden group. Conclusion The stress injury response is very strong in rabbits attacked by artillery, reverse blindage protection can reduce degree of brain tissue injury, NMDA-R1 participates in brain tissue injury pathological process.