CAJ | 학술논문

脑缺血再灌注损伤在形成和发展的过程中会引起一系列炎症因子的变化，阿托伐他汀能够通过对炎症因子直接或者间接的抑制来实现对脑缺血再灌注损伤的保护。本文对脑缺血再灌注损伤时炎症因子的免疫学机制及阿托伐他汀的保护作用进行综述，为脑缺血再灌注损伤保护作用的进一步研究提供理论依据。
뇌결혈재관주손상재형성화발전적과정중회인기일계렬염증인자적변화，아탁벌타정능구통과대염증인자직접혹자간접적억제래실현대뇌결혈재관주손상적보호。본문대뇌결혈재관주손상시염증인자적면역학궤제급아탁벌타정적보호작용진행종술，위뇌결혈재관주손상보호작용적진일보연구제공이론의거。
The cerebral ischemia reperfusion injury can cause a series of inflammatory factor ’ s change in the process of formation and development , and atorvastatin can protect cerebral ischemia reperfusion injury by inhibiting inflam -mation factor directly or indirectly .This paper reviews the immunology mechanism of inflammation factor on cere-bral ischemia reperfusion injury and atorvastatin ’ s protection , and provides theoretical basis for further study .