安徽医科大学学报
安徽醫科大學學報
안휘의과대학학보
ACTA UNIVERSITY MEDICINALIS ANHUI
2013年
12期
1542-1543,1544
,共3页
替罗非班%缺氧诱导因子-1α%高敏C反应蛋白%急性冠脉综合征
替囉非班%缺氧誘導因子-1α%高敏C反應蛋白%急性冠脈綜閤徵
체라비반%결양유도인자-1α%고민C반응단백%급성관맥종합정
tirofiban%hypoxia inducible factor-1α%high sensitivity C-reactive protein%acute coronary syndromes
将80例非ST段抬高型急性冠脉综合征(NSTE-ACS)患者随机均分为对照组和治疗组,对照组给于常规治疗,治疗组在此基础上早期即加用替罗非班治疗,分别在入院时、入院后12、24、48 h及7 d测定各组患者缺氧诱导因子-1α(HIF-1α)和高敏C反应蛋白(hs-CRP)表达水平变化。结果显示入院后两组HIF-1α水平均逐步升高,12 h左右达高峰,后逐渐下降;hs-CRP水平在入院后逐步升高,于24 h后达高峰,24 h后逐渐下降。治疗组HIF-1α和hs-CRP水平在入院后12、24、48 h和7 d均明显低于对照组(P<0.05)。
將80例非ST段抬高型急性冠脈綜閤徵(NSTE-ACS)患者隨機均分為對照組和治療組,對照組給于常規治療,治療組在此基礎上早期即加用替囉非班治療,分彆在入院時、入院後12、24、48 h及7 d測定各組患者缺氧誘導因子-1α(HIF-1α)和高敏C反應蛋白(hs-CRP)錶達水平變化。結果顯示入院後兩組HIF-1α水平均逐步升高,12 h左右達高峰,後逐漸下降;hs-CRP水平在入院後逐步升高,于24 h後達高峰,24 h後逐漸下降。治療組HIF-1α和hs-CRP水平在入院後12、24、48 h和7 d均明顯低于對照組(P<0.05)。
장80례비ST단태고형급성관맥종합정(NSTE-ACS)환자수궤균분위대조조화치료조,대조조급우상규치료,치료조재차기출상조기즉가용체라비반치료,분별재입원시、입원후12、24、48 h급7 d측정각조환자결양유도인자-1α(HIF-1α)화고민C반응단백(hs-CRP)표체수평변화。결과현시입원후량조HIF-1α수평균축보승고,12 h좌우체고봉,후축점하강;hs-CRP수평재입원후축보승고,우24 h후체고봉,24 h후축점하강。치료조HIF-1α화hs-CRP수평재입원후12、24、48 h화7 d균명현저우대조조(P<0.05)。
80 NSTE-ACS patients were randomly divided into 2 groups: the control group and the therapy group (40 cases in each group). The therapy group was treated with Tirofiban besides the conventional treatment which received by the control group. The level of HIF-1α and hs-CRP was detected immediately, 12, 24, 48 h,and 7 d after hospitalization. The HIF-1αlevel of both group increased in the first 12 hours and then decreased gradually to the baseline in 7 days. The hs-CRP level of both group increased gradually and reached its peak after 24 h after hospitalization, then decreased. Compared with the control group, the level of HIF-1α and hs-CRP decreased sig-nificantly at 12, 24, 48 h and 7 d after hospitalization in the therapy group.