CAJ | 학술논문

糖尿病认知功能障碍（cognitive impairment in diabetes， CID）是糖尿病的慢性并发症之一，其发病机制目前尚未完全清楚。近年来随着人们对CID研究的深入，发现在糖尿病整个时期，N-甲基-D-天冬氨酸受体（N-methyl-D-aspartame receptor， NMDAR）及其亚基NR2A与NR2B的变化与CID的发病机制有着密切的联系，现就此作一综述，为以后进一步研究CID打下基础。
당뇨병인지공능장애（cognitive impairment in diabetes， CID）시당뇨병적만성병발증지일，기발병궤제목전상미완전청초。근년래수착인문대CID연구적심입，발현재당뇨병정개시기，N-갑기-D-천동안산수체（N-methyl-D-aspartame receptor， NMDAR）급기아기NR2A여NR2B적변화여CID적발병궤제유착밀절적련계，현취차작일종술，위이후진일보연구CID타하기출。
Diabetic cognitive impairment is one of the chronic complications of diabetes,and the pathogenesis has not yet been fully clarified. In recent years, more and more studies of CID showed that the changes of NMDA receptor and subunits NR2A and NR2B might be important during the period of the diabetes,and they are associated with the mechanism of cognitive impairment. This article makes a summary on these researches so as to lay the foundation for a further study.