草原与草坪
草原與草坪
초원여초평
GRASSLAND AND TURF
2012年
6期
7-10,16
,共5页
NO%NaCl胁迫%燕麦%氧化损伤
NO%NaCl脅迫%燕麥%氧化損傷
NO%NaCl협박%연맥%양화손상
nitric oxide%NaC1 stress%Arena sativa%oxidative damage
以加燕2号为试验材料,用一氧化氮(NO)供体硝普钠(SNP)、NO清除剂牛血红蛋白(Hb)及SNP类似物亚铁氰化钠处理燕麦植株,研究NO对NaCl胁迫下燕麦幼苗生长和氧化损伤的缓解效应。结果表明,NaCl胁迫下添加外源NO提高了燕麦幼苗的株高,提高了SOD、POD、CAT和APX活性,降低自由基含量和膜脂过氧化程度,表明外源NO能缓解NaCl胁迫诱导的氧化损伤,增强植株的耐盐性。NO供体SNP的类似物亚铁氰化钠(不产生NO)对NaCl胁迫燕麦植株的株高和氧化伤害无缓解效应;施用Hb提高了自由基含量,降低了抗氧化酶活性,而添加外源NO能缓解Hb对燕麦生长的抑制,表明内源NO也可能参与燕麦幼苗耐盐性的调节。
以加燕2號為試驗材料,用一氧化氮(NO)供體硝普鈉(SNP)、NO清除劑牛血紅蛋白(Hb)及SNP類似物亞鐵氰化鈉處理燕麥植株,研究NO對NaCl脅迫下燕麥幼苗生長和氧化損傷的緩解效應。結果錶明,NaCl脅迫下添加外源NO提高瞭燕麥幼苗的株高,提高瞭SOD、POD、CAT和APX活性,降低自由基含量和膜脂過氧化程度,錶明外源NO能緩解NaCl脅迫誘導的氧化損傷,增彊植株的耐鹽性。NO供體SNP的類似物亞鐵氰化鈉(不產生NO)對NaCl脅迫燕麥植株的株高和氧化傷害無緩解效應;施用Hb提高瞭自由基含量,降低瞭抗氧化酶活性,而添加外源NO能緩解Hb對燕麥生長的抑製,錶明內源NO也可能參與燕麥幼苗耐鹽性的調節。
이가연2호위시험재료,용일양화담(NO)공체초보납(SNP)、NO청제제우혈홍단백(Hb)급SNP유사물아철청화납처리연맥식주,연구NO대NaCl협박하연맥유묘생장화양화손상적완해효응。결과표명,NaCl협박하첨가외원NO제고료연맥유묘적주고,제고료SOD、POD、CAT화APX활성,강저자유기함량화막지과양화정도,표명외원NO능완해NaCl협박유도적양화손상,증강식주적내염성。NO공체SNP적유사물아철청화납(불산생NO)대NaCl협박연맥식주적주고화양화상해무완해효응;시용Hb제고료자유기함량,강저료항양화매활성,이첨가외원NO능완해Hb대연맥생장적억제,표명내원NO야가능삼여연맥유묘내염성적조절。
Oats cultivars (Arena sativa cv. Jiayang No. 2) was used as material,and NO-donor SNP, NO- scavenger haemoglobin (Hb) and sodium ferrocyanide (SNP analogue that does not release NO) were used in seedling treatment to investigate the alleviative effect of NO on oxidative damage of oats under Nacl stress. The result showed that the exogenous NO increased the plant height and the activities of SOD , POD , CAT and APX,and decreased MDA and H2O2 concentrations and O2 . producing rate under salt stress. This showed that the exogenous NO can alleviate oxidative damage of oats under salt stress and enhance salt tolerance of oats. The NO-donor SNP analogue, sodium ferrocyanide (which does not release NO) had not conspicuous effect on plant height and oxidative damage of oats seedling under salt stress. The NO scavenger Hb was used to oats,the concentration of the free radicals was increased and antioxidant enzymes activity was decreased. The addition of exogenous NO ameliorated the inhibition that Hb to oats,we concluded that endogenous NO may be involved the regulation of oats seedlings for salt tolerance.