中国循环杂志
中國循環雜誌
중국순배잡지
CHINESE CIRCULATION JOURNAL
2014年
6期
440-443
,共4页
高血压%动脉硬化指数%左心室质量%左心房内径
高血壓%動脈硬化指數%左心室質量%左心房內徑
고혈압%동맥경화지수%좌심실질량%좌심방내경
Hypertension%Arterial stiffness index%Left ventricular mass index%Left atrial diameter
目的:探讨高血压患者动态动脉硬化指数(AASI)、相应对称性动态动脉硬化指数(AASI-BPVR)与左心室质量指数(LVMI)、左心房内径(LAD)的相关性。<br> 方法:采用常规24 h动态血压监测数据测定及计算286例高血压病患者AASI、AASI-BPVR以及经超声心动图测定左心室舒张末内径、室间隔厚度、左心室后壁厚度、LAD,计算左心室质量、LVMI、相对室壁厚度,同时检测空腹血糖、血脂水平,进行Pearson相关分析及多元线性回归分析。将24 h AASI>中位数(即>0.51)者为A组(n=133),24 hAASI<中位数(即≤0.51)者为B组(n=153)。<br> 结果:A组较B组LVMI增加,差异有统计学意义(P<0.05),左心室质量虽较大,但差异无统计学意义(P=0.384);经Pearson相关分析,LVMI、LAD与各动态动脉硬化指数无明显相关,其中24 h AASI-BPVR与LAD相关系数r=0.111,但差异无统计学意义(P=0.057)。<br> 结论:AASI、AASI-BPVR不是高血压左心室肥厚及左心房扩大的独立影响因素,目前尚不能作为高血压心脏损害的预测因子。
目的:探討高血壓患者動態動脈硬化指數(AASI)、相應對稱性動態動脈硬化指數(AASI-BPVR)與左心室質量指數(LVMI)、左心房內徑(LAD)的相關性。<br> 方法:採用常規24 h動態血壓鑑測數據測定及計算286例高血壓病患者AASI、AASI-BPVR以及經超聲心動圖測定左心室舒張末內徑、室間隔厚度、左心室後壁厚度、LAD,計算左心室質量、LVMI、相對室壁厚度,同時檢測空腹血糖、血脂水平,進行Pearson相關分析及多元線性迴歸分析。將24 h AASI>中位數(即>0.51)者為A組(n=133),24 hAASI<中位數(即≤0.51)者為B組(n=153)。<br> 結果:A組較B組LVMI增加,差異有統計學意義(P<0.05),左心室質量雖較大,但差異無統計學意義(P=0.384);經Pearson相關分析,LVMI、LAD與各動態動脈硬化指數無明顯相關,其中24 h AASI-BPVR與LAD相關繫數r=0.111,但差異無統計學意義(P=0.057)。<br> 結論:AASI、AASI-BPVR不是高血壓左心室肥厚及左心房擴大的獨立影響因素,目前尚不能作為高血壓心髒損害的預測因子。
목적:탐토고혈압환자동태동맥경화지수(AASI)、상응대칭성동태동맥경화지수(AASI-BPVR)여좌심실질량지수(LVMI)、좌심방내경(LAD)적상관성。<br> 방법:채용상규24 h동태혈압감측수거측정급계산286례고혈압병환자AASI、AASI-BPVR이급경초성심동도측정좌심실서장말내경、실간격후도、좌심실후벽후도、LAD,계산좌심실질량、LVMI、상대실벽후도,동시검측공복혈당、혈지수평,진행Pearson상관분석급다원선성회귀분석。장24 h AASI>중위수(즉>0.51)자위A조(n=133),24 hAASI<중위수(즉≤0.51)자위B조(n=153)。<br> 결과:A조교B조LVMI증가,차이유통계학의의(P<0.05),좌심실질량수교대,단차이무통계학의의(P=0.384);경Pearson상관분석,LVMI、LAD여각동태동맥경화지수무명현상관,기중24 h AASI-BPVR여LAD상관계수r=0.111,단차이무통계학의의(P=0.057)。<br> 결론:AASI、AASI-BPVR불시고혈압좌심실비후급좌심방확대적독립영향인소,목전상불능작위고혈압심장손해적예측인자。
Objective: To explore the relationship between ambulatory arterial stiffness indexes (AASI), AASI-blood pressure variability (AASI-BPVR) and left ventricular mass index (LVMI) left atrium diameter (LAD) in patients with hypertension. <br> Methods: A total of 286 hypertensive patients were enrolled in this study. The AASI, AASI-BPVR were calculated from 24-hour ambulatory blood pressure monitoring. Left ventricular internal dimension in diastole (LVIDd), interventricular septal thickness in diastole (IVSd), posterior wall thickness in diastole (PWd), LAD were detected by echocardiography and LVMI, relative wall thickness (RWT) were calculated. The fasting blood glucose, blood lipids were examined. According to 24 h AASI, the patients were divided into 2 groups, Group A, the patients with AASI > 0.51, n=133 and Group B, the patients with AASI ≤ 0.51,n=153. Pearson and multi regression analysis were conducted to analyze the relevant correlations. <br> Results: Group A had increased LVMI than that in Group B,P<0.05, the left ventricular masses were similar between 2 groups,P=0.384. Pearson correlation analysis indicated that LVMI and LAD were not related to arterial stiffness index, the coefifcient between 24 h-AASI and LAD was atr=0.111,P=0.057. <br> Conclusion: AASI and AASI-BPVR were not the independent factors for left ventricular hypertrophy and left atrial enlargement, therefore, they were not the predictors for cardiac damage in patients with hypertension at present time.