CAJ | 학술논문

肥胖会导致体内血浆瘦素水平升高，发生瘦素抵抗，进而影响机体的物质和能量代谢，瘦素还可直接作用于睾丸间质细胞影响睾酮的生物合成；而运动作为一种应激因素，可影响雄性生殖系统的功能。本文阐述了肥胖状态下由瘦素作用的JAK-STAT对睾酮传导通路各级蛋白的影响，以及运动干预对各级联蛋白含量的影响。
비반회도치체내혈장수소수평승고，발생수소저항，진이영향궤체적물질화능량대사，수소환가직접작용우고환간질세포영향고동적생물합성；이운동작위일충응격인소，가영향웅성생식계통적공능。본문천술료비반상태하유수소작용적JAK-STAT대고동전도통로각급단백적영향，이급운동간예대각급련단백함량적영향。
Obesity leads to elevated plasma leptin levels in the body, and produce leptin resistance, thereby affect the body's metabolism of matter and energy. Leptin can also act directly on Leydig cells and affect the biosynthesis of testosterone.Exercise as a stress factor can affect the function of the male’s reproductive system. This paper mainly discusses the influence that JAK-STAT effected by leptin exert on testosterone pathway protein under the state of obesity, as well as the influence exercise intervention laid on associated protein content at all levels.