国际脑血管病杂志
國際腦血管病雜誌
국제뇌혈관병잡지
INTERNATIONAL JOURNAL OF CEREBROVASCULAR DISEASES
2010年
1期
69-73
,共5页
运动%卒中%危险因素%高血压%糖尿病%高脂血症%颈动脉狭窄
運動%卒中%危險因素%高血壓%糖尿病%高脂血癥%頸動脈狹窄
운동%졸중%위험인소%고혈압%당뇨병%고지혈증%경동맥협착
Exercise%Stroke%Risk factors%Hypertension%Diabetes mellitus%Hyperlipidemias%Carotid stenosis
美同心脏协会/美国卒中协会的缺血性卒中一级预防指南将体力活动缺乏列为一项证据充分的可干预危险因素.以往研究表明,有氧训练可降低高血压患者或非高血压人群的血压水平,前者的血压下降更为显著;在偏瘫患者中也观察到有氧训练组收缩压显著下降.有氧运动是目前公认的治疗糖尿病的运动疗法之一.运动可提高胰岛素的敏感性,提高葡萄糖的利用率,从而降低血糖水平.超过一定阈值的运动能调节高密度脂蛋白胆固醇和三酰甘油水平,延缓颈动脉内膜中膜厚度的增加,降低高敏C-反应蛋白和血管细胞黏附分子-1水平,对内源性纤维蛋白溶解有即刻效应.因此,通过生活方式调节,特别是增加有氧运动来预防卒中已得到公认,而卒中患者能否通过积极的有氧训练达到对卒中危险因素的干预以及能否最终达到预防卒中复发的目的,还有待进一步的研究证实.
美同心髒協會/美國卒中協會的缺血性卒中一級預防指南將體力活動缺乏列為一項證據充分的可榦預危險因素.以往研究錶明,有氧訓練可降低高血壓患者或非高血壓人群的血壓水平,前者的血壓下降更為顯著;在偏癱患者中也觀察到有氧訓練組收縮壓顯著下降.有氧運動是目前公認的治療糖尿病的運動療法之一.運動可提高胰島素的敏感性,提高葡萄糖的利用率,從而降低血糖水平.超過一定閾值的運動能調節高密度脂蛋白膽固醇和三酰甘油水平,延緩頸動脈內膜中膜厚度的增加,降低高敏C-反應蛋白和血管細胞黏附分子-1水平,對內源性纖維蛋白溶解有即刻效應.因此,通過生活方式調節,特彆是增加有氧運動來預防卒中已得到公認,而卒中患者能否通過積極的有氧訓練達到對卒中危險因素的榦預以及能否最終達到預防卒中複髮的目的,還有待進一步的研究證實.
미동심장협회/미국졸중협회적결혈성졸중일급예방지남장체력활동결핍렬위일항증거충분적가간예위험인소.이왕연구표명,유양훈련가강저고혈압환자혹비고혈압인군적혈압수평,전자적혈압하강경위현저;재편탄환자중야관찰도유양훈련조수축압현저하강.유양운동시목전공인적치료당뇨병적운동요법지일.운동가제고이도소적민감성,제고포도당적이용솔,종이강저혈당수평.초과일정역치적운동능조절고밀도지단백담고순화삼선감유수평,연완경동맥내막중막후도적증가,강저고민C-반응단백화혈관세포점부분자-1수평,대내원성섬유단백용해유즉각효응.인차,통과생활방식조절,특별시증가유양운동래예방졸중이득도공인,이졸중환자능부통과적겁적유양훈련체도대졸중위험인소적간예이급능부최종체도예방졸중복발적목적,환유대진일보적연구증실.
The American Heart Association/American Stroke Association Guidelines for Primary Prevention of Stroke has listed physical inactivity as a well-documented and modifiable risk factor. Previous studies have shown that aerobic training could reduce the blood pressure levels in patients with hypertension or non-hypertensive population, especially in the former; the significantly decreased systolic pressure in patients with hemiplegia was also observed in the aerobic training group. Currently, aerobic exercise is recognized as one of the exercise therapies for diabetes. Exercise can enhance the sensitivity of insulin, and improve the utilization rate of glucose, thereby reducing the blood glucose level. Exercise over a certain threshold can regulate the levels of high-density lipoprotein cholesterol and triglyceride, delay the increased carotid artery intima-media thickness, reduce the levels of high-sensitivity C-reactive protein and vascular cell adhesion molecule-1, and have immediate effects on endogenous fibrinolysis. Therefore, by regulating lifestyle, particularly by increasing aerobic exercise to prevent stroke has been recognized, and whether the patients with stroke can achieve the intervention of the risk factors for stroke by the active aerobic training, as well as whether they can ultimately achieve the purpose of preventing stroke recurrence are still need to be confirmed by further studies.
