CAJ | 학술논문

目的观察高压舱压力变化对舱内人员眼压的影响,并探讨其发生机制.方法健康志愿者17人34眼,男13人,女4人,年龄18 ～ 34岁,平均(25.76±5.16)岁.在检查者和被检查者均不了解研究目的的情况下,被检查者进入高压舱,检查者用日本产NIDEK NT-2000非接触性眼压计分别测量被检查者下列状态下的眼压:安静状态下(初始眼压)、常压下不吸纯氧停留30 min时、常压下吸纯氧10 min后、不吸纯氧以2.0 kPa/s加压至60 kPa时、不吸纯氧60 kPa停留30 min时、不吸纯氧压力减至0 kPa时.结果不吸纯氧常压下停留30 min时、常压下吸纯氧10 min后、不吸纯氧压力减至0 kPa时的平均眼压分别为(13.57±3.04)、(13.86±3.16)、(13.33 ±-3.12) mmHg,与基础眼压[(13.48±2.87) mmHg]比较差异均无统计学意义(P＞0.05);不吸纯氧加压至60 kPa时、不吸纯氧60 kPa停留30 min时的平均眼压分别为(16.06±2.48)、(15.65±2.54) mmHg,与基础眼压比较差异均有统计学意义(P＜0.01).结论当环境压力突然增加时,多数人会出现眼压调节不良性升高,少数人出现眼压调节不良性降低,也有少数人的眼压基本保持不变.
목적 관찰고압창압력변화대창내인원안압적영향,병탐토기발생궤제.방법 건강지원자17인34안,남13인,녀4인,년령18 ～ 34세,평균(25.76±5.16)세.재검사자화피검사자균불료해연구목적적정황하,피검사자진입고압창,검사자용일본산NIDEK NT-2000비접촉성안압계분별측량피검사자하렬상태하적안압:안정상태하(초시안압)、상압하불흡순양정류30 min시、상압하흡순양10 min후、불흡순양이2.0 kPa/s가압지60 kPa시、불흡순양60 kPa정류30 min시、불흡순양압력감지0 kPa시.결과 불흡순양상압하정류30 min시、상압하흡순양10 min후、불흡순양압력감지0 kPa시적평균안압분별위(13.57±3.04)、(13.86±3.16)、(13.33 ±-3.12) mmHg,여기출안압[(13.48±2.87) mmHg]비교차이균무통계학의의(P＞0.05);불흡순양가압지60 kPa시、불흡순양60 kPa정류30 min시적평균안압분별위(16.06±2.48)、(15.65±2.54) mmHg,여기출안압비교차이균유통계학의의(P＜0.01).결론 당배경압력돌연증가시,다수인회출현안압조절불량성승고,소수인출현안압조절불량성강저,야유소수인적안압기본보지불변.
Objective To observe whether pressure boost could induce changes in intracardiac pressures,and also to investigate the mechanism involved.Methods Seventeen healthy volunteers [13 males and 4 females,with an age range of 25.76 ± 5.16 years,and average age of 18-34] were recruited for the study.Without the knowledge of the examiners and examinees about the purpose of the research,they entered the hyperbaric chamber.The examiners detect the intraocular pressures (IOP) of the examinees with the Japanese NIDEK NT-2000 non-contact tonometer under the following conditions:(1) baseline intraocular pressures at rest.(2) during the stay at atmospheric pressure for 30 min without breathing pure oxygen; after breathing pure oxygen for 10 min at atmospheric pressure.(3) compression to a pressure of 60 kPa at a rate of 2.0 kPa/s without breathing pure oxygen; (4) during the stay at the pressure of 60 kPa for 30 min without breathing pure oxygen ; (5) after decompression to 0 kPa pressure,also without breathing pure oxygen.Results Average intraocular pressures measured during their stay at atmospheric pressure for 30 min without breathing pure oxygen,after breathing pure oxygen for 10 min at atmospheric pressure and after decompression to 0 kPa pressure without breathing pure oxygen were (13.57 ±3.04),(13.86 ± 3.16) and (13.33 ± 3.12) mmHg respectively.No statistical significance could be seen in measured data,as compared with that of the baseline intraocular pressures [(13.48 ± 2.87) mmHg] (P ＞ 0.05).Average intraocular pressures measured after they were compressed to 60 kPa without breathing pure oxygen and during their stay at 60 kPa for 30 min without breathing pure oxygen were (16.06 ±2.48) and (15.65 ± 2.54) mmHg respectively,and statistical difference could be seen,when compared with that of the baseline intraocular pressures(P ＞ 0.01).Conclusions When there was a pressure boost,most people would display elevated IOP due to poor response to pressure,only a few people would experience IOP drop also due to poor response to pressure and IOP of still a few people would remain basically unchanged.