CAJ | 학술논문

目的探讨永存三叉动脉(PTA)合并脑血管病的脑循环及临床特征. 方法选择自2010年3月至2013年3月第二炮兵总医院神经内科和神经介入科收治的经数字减影血管造影(DSA)、磁共振血管造影(MRA)或CT血管成像(CTA)证实存在PTA的脑血管病患者8例为研究对象,回顾性分析其脑循环特征及临床表现. 结果 8例PTA合并脑血管病患者中,2例合并脑梗死,1例合并脑动脉狭窄,2例合并脑梗死和脑动脉狭窄,1例合并短暂性脑缺血发作,2例合并动脉瘤;发作性头晕4例(其中1例伴一侧肢体力弱),一侧肢体麻木2例,头痛、眼睑下垂1例,复视1例;PTA分型Saltzman Ⅰ型6例,SaltzmanⅡ型1例,特殊类型1例.4例脑梗死的发病机制均与PTA相关. 结论 PTA合并脑血管病患者的临床表现多由其合并疾病所决定.PTA的存在可改变正常脑循环途径,对其进行检测可为多发性脑梗死发病机制的分析提供帮助.
목적 탐토영존삼차동맥(PTA)합병뇌혈관병적뇌순배급림상특정. 방법 선택자2010년3월지2013년3월제이포병총의원신경내과화신경개입과수치적경수자감영혈관조영(DSA)、자공진혈관조영(MRA)혹CT혈관성상(CTA)증실존재PTA적뇌혈관병환자8례위연구대상,회고성분석기뇌순배특정급림상표현. 결과 8례PTA합병뇌혈관병환자중,2례합병뇌경사,1례합병뇌동맥협착,2례합병뇌경사화뇌동맥협착,1례합병단잠성뇌결혈발작,2례합병동맥류;발작성두훈4례(기중1례반일측지체력약),일측지체마목2례,두통、안검하수1례,복시1례;PTA분형Saltzman Ⅰ형6례,SaltzmanⅡ형1례,특수류형1례.4례뇌경사적발병궤제균여PTA상관. 결론 PTA합병뇌혈관병환자적림상표현다유기합병질병소결정.PTA적존재가개변정상뇌순배도경,대기진행검측가위다발성뇌경사발병궤제적분석제공방조.
Objective To explore the cerebral circulation characteristics of patients with persistent trigeminal artery (PTA) combined with cerebrovascular diseases and their clinical relevance.Methods Eight patients with PTA,admitted our hospital from March 2010 to March 2013 and conformed by DSA,MR radiography (MRA) or CT angiography (CTA),were chosen in our study; their cerebral circulation features and clinical manifestations were retrospectively analyzed.Results In these eight patients,two were combined with cerebral infarction,one with cerebral artery stenosis,two with cerebral infarction and cerebral artery stenosis,one with transient ischemic attack,and the left two with cerebral aneurysm.Four patients complained for paroxysmal dizziness (one of them with one-side weakness),two patients complained for numbness of a limb,One patient had headache and blepharoptosis and one patient had diplopia.Saltzman type Ⅰ was noted in six patients,type H in one and special type in one.The pathogenesis of cerebral infarction was related to PTA.Conclusion The clinical features of the patients with PTA often determine by merger cerebrovascular diseases; PTA can change the normal cerebral circulation; PTA detection can be helpful in the diagnosis of the pathogenesis of multiple cerebral infarction.