世界最新医学信息文摘(连续型电子期刊)
世界最新醫學信息文摘(連續型電子期刊)
세계최신의학신식문적(련속형전자기간)
World Latest Medicine Information
2014年
31期
32-33
,共2页
计小刚%单保安%王同山%丁亚平
計小剛%單保安%王同山%丁亞平
계소강%단보안%왕동산%정아평
肠梗阻%临床分析%药物疗法
腸梗阻%臨床分析%藥物療法
장경조%림상분석%약물요법
intestinal obstruction%clinical analysis%drug therapy
目的:初步探讨术后早期炎性肠梗阻临床特点及治疗措施。方法选择我院1997年至2005年诊治的8例炎性肠梗阻进行临床回顾性分析,并进行总结。结果经治疗后肠梗阻症状在6~30d 解除。两例二次手术一例造成肠瘘,本组无死亡,无再手术,均治愈出院。结论术后早期炎性肠梗阻是由小肠、结肠间浸润的白细胞介导的炎症反应引起;腹部手术创伤或腹腔内炎性反应等因素导致肠壁水肿,渗出增加,引起白细胞聚集,诱发腹腔炎症反应,引起肠管间麻痹、炎性反应、水肿和粘连,最后导致肠梗阻,其中炎性反应造成的肠蠕动减弱是梗阻的主要原因。预防及治疗主要是针对减轻炎症反应减少炎症介质释放。
目的:初步探討術後早期炎性腸梗阻臨床特點及治療措施。方法選擇我院1997年至2005年診治的8例炎性腸梗阻進行臨床迴顧性分析,併進行總結。結果經治療後腸梗阻癥狀在6~30d 解除。兩例二次手術一例造成腸瘺,本組無死亡,無再手術,均治愈齣院。結論術後早期炎性腸梗阻是由小腸、結腸間浸潤的白細胞介導的炎癥反應引起;腹部手術創傷或腹腔內炎性反應等因素導緻腸壁水腫,滲齣增加,引起白細胞聚集,誘髮腹腔炎癥反應,引起腸管間痳痺、炎性反應、水腫和粘連,最後導緻腸梗阻,其中炎性反應造成的腸蠕動減弱是梗阻的主要原因。預防及治療主要是針對減輕炎癥反應減少炎癥介質釋放。
목적:초보탐토술후조기염성장경조림상특점급치료조시。방법선택아원1997년지2005년진치적8례염성장경조진행림상회고성분석,병진행총결。결과경치료후장경조증상재6~30d 해제。량례이차수술일례조성장루,본조무사망,무재수술,균치유출원。결론술후조기염성장경조시유소장、결장간침윤적백세포개도적염증반응인기;복부수술창상혹복강내염성반응등인소도치장벽수종,삼출증가,인기백세포취집,유발복강염증반응,인기장관간마비、염성반응、수종화점련,최후도치장경조,기중염성반응조성적장연동감약시경조적주요원인。예방급치료주요시침대감경염증반응감소염증개질석방。
Objective to investigate the clinical features and treatment measures of early postoperative inflammatory intestinal obstruction.Methods The clinical data of eight cases of patients with inflammatory intestinal obstruction admitted in our hospital from 1997 to 2005 were retrospectively analyzed and summarized.Results after treatment, the symptoms of intestinal obstruction were lifted in 6d to 30d. Two cases were treated by reoperation resulting in intestinal fistula in one case. there were no death cases and they didn’t need further surgery. they were cured. Conclusions early postoperative inflammatory intestinal obstruction was caused by inflammatory reaction mediated by infiltrating leukocytes between the small intestine and colon. The abdominal surgery, trauma or intra-abdominal inflammation and other factors led to the intestinal wall edema, increasing exudation, resulting in leukocyte aggregation which induced peritoneal inflammation, thus causing paralysis between bowel, inflammation, swelling and adhesions, as a result intestinal obstruction occurred. The inflammation induced peristalsis weaken is the main cause of intestinal obstruction. What we aimed at for prevention and treatment in intestinal obstruction was mainly reducing the release of inflammatory mediators to reduce inflammation.