CAJ | 학술논문

以典型抗癫痫药物卡马西平为目标污染物，研究热活化过硫酸盐（thermally activated persulfate，TAP）技术对其的降解效果。此外，还考察了过硫酸盐初始浓度、温度和零价铁投加量等对降解效果的影响。结果表明，随着过硫酸盐初始浓度的增加，降解速率常数提高，不同温度下卡马西平降解速率常数与过硫酸盐初始浓度表现出良好的线性关系。提高系统温度能够提高卡马西平的降解速率。TAP氧化卡马西平符合拟一级动力学，反应活化能Ea为(120.4±2.6)kJ·mol?1。在TAP系统中加入少量零价铁能够显著地提高卡马西平的降解速率和矿化度。当温度为60℃时，零价铁的最佳投加量为0.05 g·L?1。硫酸自由基易于对卡马西平分子结构中氮杂卓环的烯烃双键进行攻击，主要生成羟基化卡马西平、环氧卡马西平、吡啶类醛和酮等中间产物。
이전형항전간약물잡마서평위목표오염물，연구열활화과류산염（thermally activated persulfate，TAP）기술대기적강해효과。차외，환고찰료과류산염초시농도、온도화령개철투가량등대강해효과적영향。결과표명，수착과류산염초시농도적증가，강해속솔상수제고，불동온도하잡마서평강해속솔상수여과류산염초시농도표현출량호적선성관계。제고계통온도능구제고잡마서평적강해속솔。TAP양화잡마서평부합의일급동역학，반응활화능Ea위(120.4±2.6)kJ·mol?1。재TAP계통중가입소량령개철능구현저지제고잡마서평적강해속솔화광화도。당온도위60℃시，령개철적최가투가량위0.05 g·L?1。류산자유기역우대잡마서평분자결구중담잡탁배적희경쌍건진행공격，주요생성간기화잡마서평、배양잡마서평、필정류철화동등중간산물。
The antiepileptic drug carbamazepine (CBZ) was chosen as the target compound to investigate the degradation efficiency of an oxidation process by thermally activated persulfate (TAP). Furthermore, some key factors affecting this efficiency such as initial persulfate concentrations, temperature and zero-valent iron dosage were also evaluated. The resulted showed that the rate constant of degradating CBZ increased with increase of the initial persulfate concentrations. There appeared a linear relation between the degradation rate constant and the initial persulfate concentration. Elevating temperature could improve the carbamazepine degradation. Carbamazepine degradation by TAP oxidation abided by pseudo-first-order kinetics, activation energy (Ea) was (120.4±2.6) kJ·mol?1. The degradation rate and mineralization degree for Carbamazepine can be significantly improved if a small amount of zero-valent iron was added into TAP system. When the system temperature set at 60℃, the optimum dosage of zero-valent iron was 0.05 g·L?1. Sulfate radicals were prone to attack alkene double bonds in azepine ring and the main intermediate products found were hydroxycarbamazepine, epoxycarbamazepine, pyridine aldehydes and ketones.