国际眼科杂志
國際眼科雜誌
국제안과잡지
INTERNATIONAL JOURNAL OF OPHTHALMOLOGY
2015年
2期
307-310
,共4页
牟章兵%向金梅%黄旭%仁千格麦%伍晓辉%彭引
牟章兵%嚮金梅%黃旭%仁韆格麥%伍曉輝%彭引
모장병%향금매%황욱%인천격맥%오효휘%팽인
准分子激光原位角膜磨镶术%并发症%治疗
準分子激光原位角膜磨鑲術%併髮癥%治療
준분자격광원위각막마양술%병발증%치료
laser in situ keratomileusis%complication%treatment
目的：：回顾性分析准分子激光原位角膜磨镶术( LASIK)术后视力下降的原因,并初步探讨处理和预防措施。方法：收集LASIK术后视力下降病例134例175眼,统计各种原因的构成比,分别将治疗前后的UCVA进行配对t检验。结果：LASIK术后视力下降的总体发生率为1.86%,其中屈光回退的构成比为51.43%,治疗后 UCVA 由0.61±0.22上升为0.90±0.38(t=8.00,P<0.001)；角膜上皮点状缺损构成比为32.57%,治疗后UCVA由0.60±0.19上升为1.20±0.24(t=20.00,P<0.001)；调节痉挛构成比为5.14%,治疗后UCVA由0.76±0.21上升为1.32±0.22(t=8.14,P<0.001)；角膜瓣移位和皱褶构成比为4%,治疗后UCVA由0.29 ± 0.26上升为1.24 ± 0.28( t=6.33, P<0.001)；激素性高眼压构成比为4%,治疗后 UCVA 由0.57±0.05上升为1.0±0.16(t=2.53,P<0.05)；眼底病变和弥漫性板层角膜炎( diffuse lamellar keratitis, DLK)构成比共为2.86%,经治疗后UCVA均有不同程度提高。结论：LASIK术后视力下降的原因多种多样,及时发现和处理可获得良好疗效,恰当的预防措施也必不可少。
目的：：迴顧性分析準分子激光原位角膜磨鑲術( LASIK)術後視力下降的原因,併初步探討處理和預防措施。方法：收集LASIK術後視力下降病例134例175眼,統計各種原因的構成比,分彆將治療前後的UCVA進行配對t檢驗。結果：LASIK術後視力下降的總體髮生率為1.86%,其中屈光迴退的構成比為51.43%,治療後 UCVA 由0.61±0.22上升為0.90±0.38(t=8.00,P<0.001)；角膜上皮點狀缺損構成比為32.57%,治療後UCVA由0.60±0.19上升為1.20±0.24(t=20.00,P<0.001)；調節痙攣構成比為5.14%,治療後UCVA由0.76±0.21上升為1.32±0.22(t=8.14,P<0.001)；角膜瓣移位和皺褶構成比為4%,治療後UCVA由0.29 ± 0.26上升為1.24 ± 0.28( t=6.33, P<0.001)；激素性高眼壓構成比為4%,治療後 UCVA 由0.57±0.05上升為1.0±0.16(t=2.53,P<0.05)；眼底病變和瀰漫性闆層角膜炎( diffuse lamellar keratitis, DLK)構成比共為2.86%,經治療後UCVA均有不同程度提高。結論：LASIK術後視力下降的原因多種多樣,及時髮現和處理可穫得良好療效,恰噹的預防措施也必不可少。
목적：：회고성분석준분자격광원위각막마양술( LASIK)술후시력하강적원인,병초보탐토처리화예방조시。방법：수집LASIK술후시력하강병례134례175안,통계각충원인적구성비,분별장치료전후적UCVA진행배대t검험。결과：LASIK술후시력하강적총체발생솔위1.86%,기중굴광회퇴적구성비위51.43%,치료후 UCVA 유0.61±0.22상승위0.90±0.38(t=8.00,P<0.001)；각막상피점상결손구성비위32.57%,치료후UCVA유0.60±0.19상승위1.20±0.24(t=20.00,P<0.001)；조절경련구성비위5.14%,치료후UCVA유0.76±0.21상승위1.32±0.22(t=8.14,P<0.001)；각막판이위화추습구성비위4%,치료후UCVA유0.29 ± 0.26상승위1.24 ± 0.28( t=6.33, P<0.001)；격소성고안압구성비위4%,치료후 UCVA 유0.57±0.05상승위1.0±0.16(t=2.53,P<0.05)；안저병변화미만성판층각막염( diffuse lamellar keratitis, DLK)구성비공위2.86%,경치료후UCVA균유불동정도제고。결론：LASIK술후시력하강적원인다충다양,급시발현화처리가획득량호료효,흡당적예방조시야필불가소。
To analyze the reasons of impaired vision after LASlK and explore its preventive treatment measures preliminarily.METHODS: ln this retrospective study, 175 eyes of 134 patients whose vision was decreased after LASlK were included. The constituent ratio of every reason was counted and uncorrected visual acuity ( UCVA ) between pre-treatment and post-treatment were compared by paired t-test respectively.RESULTS:The overall incidence of impaired vision after LASlK was 1. 86%. The constituent ratio of regression was 51. 43% and UCVA increased from 0. 61±0. 22 to 0. 90±0. 38 (t=8. 00, P<0. 001) after treatment. The constituent ratio of punctate corneal epithelial defect was 32. 57% and UCVA increased from 0. 60±0. 19 to 1. 20±0. 24 (t=20. 00, P<0. 001 ) after treatment. The constituent ratio of accommodative spasm was 5. 14% and UCVA increased from 0.76±0. 21 to 1. 32±0. 22 (t=8. 14, P<0. 001) after treatment. The constituent ratio of corneal flap shift and gauffer was 4% and UCVA increased from 0. 29 ± 0. 26 to 1. 24 ± 0. 28 ( t = 6. 33, P<0. 001 ) after treatment. The constituent ratio of corticosteroid - induced ocular hypertension was 4% and UCVA increased from 0. 57±0. 05 to 1. 0 ± 0. 16 ( t= 2. 53, P<0. 05 ) after treatment. The constituent ratio of fundus lesions and diffuse lamellar keratitis ( DLK) was 2. 86% and UCVA all increased by different degrees after treatment.CONCLUSlON: The reasons of impaired vision after LASlK are many and varied. These cases could recover their vision by discovery and treatment in time, and the appropriate preventive measures were essential.
