CAJ | 학술논문

目的：讨论脑胶质瘤致癫痫的几种因素。方法回顾性分析85例经神经影像学和病理学检查确诊为脑胶质瘤患者的病例资料。结果在85例脑胶质瘤患者中40例(47.1%)伴发癫痫。肿瘤主体位于额叶、颞叶，具有钙化、肿瘤累及皮层和无明显占位效应的胶质瘤容易引发癫痫，低级别胶质瘤患者癫痫发病率高于高级别胶质瘤患者(P<0.05)。结论脑胶质瘤所致癫痫与其病理类型、肿瘤生长部位及特征有关。
목적：토론뇌효질류치전간적궤충인소。방법회고성분석85례경신경영상학화병이학검사학진위뇌효질류환자적병례자료。결과재85례뇌효질류환자중40례(47.1%)반발전간。종류주체위우액협、섭협，구유개화、종류루급피층화무명현점위효응적효질류용역인발전간，저급별효질류환자전간발병솔고우고급별효질류환자(P<0.05)。결론뇌효질류소치전간여기병리류형、종류생장부위급특정유관。
Objective To discuss several factors in epilepsy induced by brain glioma.Methods A retrospective analysis were made on the clinical data of 85 patients with brain glioma, who were diagnosed by neuroimaging and pathology examination.Results Among the 85 patients with brain glioma, there were 40 cases (47.1%) complicated with epilepsy. Epilepsy could be easily induced by tumor in frontal lobe and temporal lobe with calcification, involved cortex, and non-obvious mass effect. Morbidity of epilepsy in low-level glioma patients was higher than that in high-level ones (P<0.05).Conclusion Epilepsy induced by brain glioma is related with its pathological pattern, tumor position and characteristics.