临床肺科杂志
臨床肺科雜誌
림상폐과잡지
JOUNAL OF CLINICAL PULMONARY MEDICINE
2015年
6期
990-992
,共3页
结核性胸膜炎%恶性胸水%内科胸腔镜%经皮穿刺胸膜盲检%确诊率
結覈性胸膜炎%噁性胸水%內科胸腔鏡%經皮穿刺胸膜盲檢%確診率
결핵성흉막염%악성흉수%내과흉강경%경피천자흉막맹검%학진솔
tuberculous pleuritis%malignant pleural effusion%internal medicine thoracoscope%percutaneous puncture biopsy of pleura%diagnosis rate
目的：研究经皮穿刺胸膜盲检对结核性胸膜炎及恶性胸水确诊率差异的原因。方法回顾性分析结核性胸膜炎20例、恶性胸水12例胸腔镜检查结果，观察两组患者镜下形态学差异，分析胸膜结节或白斑样病变间黏膜组织的活检病理结果；结果肿瘤与结核均以弥漫性分布、结节状病变为主。在病变之间的胸膜组织也均呈现出充血、水肿、增厚、粗糙、纤维粘连等异常的形态特点。对病变间黏膜组织进行活检的结果提示：结核性胸膜炎组(95%)病理符合率显著高于恶性胸水组(16．7%)(P<0．01)。结论结核特征性病变分布更广泛，是导致经皮穿刺胸膜盲检对结核、恶性胸水确诊率差异的主要原因。
目的：研究經皮穿刺胸膜盲檢對結覈性胸膜炎及噁性胸水確診率差異的原因。方法迴顧性分析結覈性胸膜炎20例、噁性胸水12例胸腔鏡檢查結果，觀察兩組患者鏡下形態學差異，分析胸膜結節或白斑樣病變間黏膜組織的活檢病理結果；結果腫瘤與結覈均以瀰漫性分佈、結節狀病變為主。在病變之間的胸膜組織也均呈現齣充血、水腫、增厚、粗糙、纖維粘連等異常的形態特點。對病變間黏膜組織進行活檢的結果提示：結覈性胸膜炎組(95%)病理符閤率顯著高于噁性胸水組(16．7%)(P<0．01)。結論結覈特徵性病變分佈更廣汎，是導緻經皮穿刺胸膜盲檢對結覈、噁性胸水確診率差異的主要原因。
목적：연구경피천자흉막맹검대결핵성흉막염급악성흉수학진솔차이적원인。방법회고성분석결핵성흉막염20례、악성흉수12례흉강경검사결과，관찰량조환자경하형태학차이，분석흉막결절혹백반양병변간점막조직적활검병리결과；결과종류여결핵균이미만성분포、결절상병변위주。재병변지간적흉막조직야균정현출충혈、수종、증후、조조、섬유점련등이상적형태특점。대병변간점막조직진행활검적결과제시：결핵성흉막염조(95%)병리부합솔현저고우악성흉수조(16．7%)(P<0．01)。결론결핵특정성병변분포경엄범，시도치경피천자흉막맹검대결핵、악성흉수학진솔차이적주요원인。
Objective To analyze the cause of different diagnostic value to tuberculous pleuritis and malig-nant pleural effusion by percutaneous puncture biopsy. Methods The internal medicine thoracoscope results of 20 tuberculous pleuritis cases and 12 malignant pleural effusion cases were retrospectively analyzed. The difference of morphology manifestation was compared between the two groups. The pathological results of pleura biopsy between le-sions were also analyzed. Results Both malignant and tuberculous groups showed nodular and diffused lesions. The mucosal tissue between nodular lesions mainly showed abnormal appearance, such as congestion, edema, thickening, rough, fibrous adhesions and so on. The characteristic pathology results could be find in 95% cases for tuberculous pleuritis, while 16. 7% in malignant pleural effusion group (P<0. 05). Conclusion Different diagnostic value of percutaneous puncture biopsy to tuberculous and malignant pleuritis could be explained by wider distribution of tuber-culous characteristic lesion.
