CAJ | 학술논문

肝硬化失代偿期出现腹水的病理生理学机制将导致心率增快、血容量及心输出量增加、外周血管阻力和平均动脉压下降的血流动力学改变并形成高动力循环状态，结合现代技术对脉象的研究，当机体处于高动力循环状态时将符合洪脉的血流动力学特点，这与目前学界公认的肝硬化腹水（即“臌胀”病）各证型脉象存在较大差异，发现此一差异对“臌胀”病的证候诊断的现代化、客观化和规范化建设有一定参考作用。
간경화실대상기출현복수적병리생이학궤제장도치심솔증쾌、혈용량급심수출량증가、외주혈관조력화평균동맥압하강적혈류동역학개변병형성고동력순배상태，결합현대기술대맥상적연구，당궤체처우고동력순배상태시장부합홍맥적혈류동역학특점，저여목전학계공인적간경화복수（즉“고창”병）각증형맥상존재교대차이，발현차일차이대“고창”병적증후진단적현대화、객관화화규범화건설유일정삼고작용。
Objective The pathophysiologic mechanism of decompensated liver cirrhosis with ascites will lead to increased heart rate, blood volume, cardiac output and decreased peripheral vascular resistance and mean arterial pressure, which form the hyperdynamic circulation. Combining with modern technology, the research on the TCM pulse manifestation shows that when the body is in hyperdynamic circulation the hemodynamic feature accord with that performs in Surging pulse.There is a big difference between the common view of pulses manifest in various syndrome types of decompensated liver cirrhosis with ascites ( ie“tympanites” disease) .It provides reference while in establishing the modernized, objective and standardized syndrome diagnosis of tympanites disease.